Research
Modified cytokinin forms can retain leaf performance in salt stress senescence
Using multi-omics analysis, we identify the function of modified cytokinin forms towards regulating stress-induced leaf senescence. For this project, I explore physiological efficiency changes under senescence progression along with an integrated transcriptomics and proteomics approach to identify regulation of genes and proteins by cytokinin. Further, using WGCNA cytokinin-specific modules were identified. The work sheds light on the action of trans-Zeatin N-glucoside forms towards altering cytokinin metabolism and photosynthesis.
Mechanism of cytokinin regulation of chloroplast function
Cytokinin plays a major role in regulating chloroplast biogenesis and division. Here we explore genes involved in chloroplast assembly as downstream targets of cytokinin action. Phenotypic characterization of chloroplast-related genes informs towards regulation of senescence onset and progression.
Source to sink transition mediated by cytokinin
Cytokinin regulates source to sink transition facilitating younger leaf development. This project explores action of cytokinin towards sugar transporters involved in sucrose transport and phloem loading. Using physiological characterization of loss-of-function mutants, this project explores sugar accumulation and whole-plant development alterations.
Published articles (Selective list)
Link to all articles: Google Scholar
Khanna RR, Hasannin O, Thennakoon M, Buchheit A, PetÅ™ík I, Strnad M, Novák O, ÄŒerný M, & Rashotte AM (2025). trans-Zeatin N-glucosides Can Delay Salt Accelerated Leaf Senescence in Arabidopsis thaliana. Plant Physiology and Biochemistry, 110101.
